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Using intestinal organoid treated with filtered fecal supernatants from term or pre-term infants, Dougherty and team demonstrated that microbial metabolites can promote enterocyte proliferation and maturation 16

This extracellular and mislocalized intracellular copper exacerbates DCM through multiple pathways, activating the TGF-/Smad signaling pathway and amplifying oxidative stress, collectively promoting extracellular matrix deposition and fibrotic remodeling [171]

Collectively, these findings indicate that SIRT1 is essential for the maintenance of redox balance and mitochondrial function in the cochlea after traumatic noise exposure, thus providing a promising therapeutic target for NIHL treatment