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Ryu HJ, Kim C, Jang H, Kim Sun II, Shin SJ, Torres-Cabala CA , Kim SK

Supplemented free-form BCAAs may cause a sharp rise in circulating BCAA levels, potentially interfering with the transport of aromatic amino acids across the bloodbrain barrier

ATF4, a transcriptional activator of ASNS, is upregulated when TP53 deleted, leading to increased ASNS expression and increased intracellular asparagine synthesis ( GLS1 (GAC, glutaminase C), such as CB-839 and DONs, suppress glutamine metabolism, mainly by affecting GDH in the active site of GAC reducing its conversion to glutamate and thus limiting the synthesis of asparagine