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The paracrine activation of GABA A receptors on NSC populations by their progeny has been shown to have a non-synaptic inhibitory effect on proliferation and likely function as a negative feedback mechanism to modulated proliferation in adult and post-natal brains [120, 121]

D., Acharya, M

The chart ranks the most-promoted ones by their demonstrated effect on the brain

The c-MET serves as an attractive therapeutic target in SCLC, as shown through small interfering RNA (siRNA) and selective prototype c-MET inhibitor SU11274, inhibiting the phosphorylation of c-MET itself and its downstream molecules such as AKT, S6 kinase, and ERK1/2