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Indeed, recent data suggest that a chronic elevation of homocysteine level might indirectly reduce methylation of DNA, RNA, proteins, and phospholipids through a concomitant increase in SAH level secondary to homocysteine-mediated reversal of the SAH hydrolase reaction (Yi et al, 2000)

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All around mitochondrial health and homeostasis under BC treatments The subjective experience of fatigue, a common symptom during the post-treatment phase of various oncological therapies, can be linked at the cellular level to the functioning of the energy metabolism, with mitochondria being the central players in this process