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Fibroblast proliferation leads to cardiac fibrosis, which replaces injured cardiomyocytes but excessive fibrosis leads to structural deformation, reduced heart function, ventricular enlargement, cardiomyocyte hypertrophy, increased wall stiffness, and progression to heart failure [7]

Conversely, boosting NAD+ levels has been proposed as a potential protective strategy against oxidative damage (Xie et al., 2020)

In fact, we encourage it

For additional insights, we performed all-atom molecular dynamics (MD) simulations for the two adjacent subunits constituting the active site from upper and lower hexamers of Pf GS and I 2 Pf GS