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Recent studies of OA cartilage homeostasis have focused on the increase in senescent cells in articular cartilage [4,5,6] can result from mitochondrial dysfunction, excessive mechanical loading, oxidative stress, and DNA damage [7,8,9,10]

Cellular regulation by hydrogen peroxide

Biochemical studies of three Saccharomyces cerevisiae acyl-CoA synthetases, Faa1p, Faa2p, and Faa3p

While microglial signaling has been reported to influence astrocytic transporter regulation, the astrocyte-enriched nature of our cultures suggests that the observed changes in GLT-1 trafficking largely reflect astrocytic responses to ischemic stress