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All these patients had defects in DMEs genes and also harbored T315I kinase domain mutation already associated with nilotinib non-responsiveness in CML patients [6]

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C.EtgenA

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Sensing of physical extracellular cues and the subsequent dynamics of the interaction between cells and the ECM regulate the downstream mechanotransductive events, causing a variety of nano- and micro-topography-sensitive cellular behaviors, including cell adhesion, morphology, proliferation, gene expression, self-renewal, and differentiation (Lemischka and Moore, 2003

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Binding of the drug to plasma proteins is variable

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