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Finally, searching for biomarkers to predict the efficiency of glutamine targeting, or metabolomic and proteomic approaches to identify metabolic compensation mechanisms, will significantly improve the efficacy of cancer treatment

These results suggest that the principle of desensitization obtained from this study of D2-like receptorsthat is, deubiquitination of arrestins via USP33 and nuclear entry of the G/arrestin complex into the nucleusalso applies to the desensitization of 2 AR

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[46] It should, however, be noted that although the two enzymes catalyze similar reactions, these generally occur in opposite directions: dihydrolipoyl dehydrogenase uses NAD + to oxidize two SH groups to a disulfide (SS)