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Biotransformation Antiarrhythmic drugs (AADs) undergo metabolism in the liver through CYP450 isoenzymes, resulting in active metabolites that either block sodium (Na+) channels (such as mexiletine and propafenone), extend action potential duration (APD) [for instance, N-acetylprocainamide (NAPA)], or cause central nervous system (CNS) toxicity (as seen with lidocaine) (11)

Deficits in cobalamin (B12), a cobalt-containing coordination molecule, can lead to megaloblastic anemia and neurological disorders

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(Low Wangs views do not represent those of the FDA or the FDA Endocrinologic and Metabolic Drugs Advisory Committee.) How much weight can people lose with retatrutide