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Description

Our approach to identify pathways directly affected by Gsta4 over-expression and sequent reduction of mitochondrial 4-HNE load consisted in overlapping two independent databases of 4-HNE modified proteins, together with assessing their transcription in the OL lineage

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This effect was exacerbated by lack of Nrf2 , indicating the importance of redox homeostasis in erythropoiesis, where Nrf2 and selenoproteins may play a synergistic role [17]