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A few DPI complexes (e.g., DPI7, DPI12, and DPI17) share the same binding site with RSL3 and act as inducers of ferroptosis (Yang et al., 2016)

reported its potential anti-fibrosis function by attenuating bleomycin-induced thickened alveolar septa, accumulation of inflammatory cells and foci of fibrotic consolidation (65), this may be related to the reduction of lung prolyl hydroxylase activity since the increased activity of this enzyme is positively correlated with and preferentially expressed in the accumulation of collagen in lungs (66)

The dose required, the side effects, the long-term safety, all are unknown

Highest yields were obtained with BtzSH ( 36 ) and BoxSH ( 37 )