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in 2015 exposes a possible cancer-stimulating eect of anti- oxidants, particularly NAC, that, as stated above, represents a GSH precursor, on in vivo and in vitro melanoma models

HFD impairs liver glutathione biosynthesis, vitamin D metabolism genes and genes associated with non-alcoholic fatty liver disease (NAFLD) Genes involved in the GSH biosynthesis pathway were significantly downregulated in the livers of mice fed an HFD compared to those of mice fed a healthy diet (controls) (Fig

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Ferroptosis: A Regulated Cell Death Nexus Linking Metabolism, Redox Biology, and Disease