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BRAF inhibitor-induced MAPK suppression triggers paradoxical ECM remodeling via mesenchymal dedifferentiation programs, activating YAP/MRTF mechanosensors that sustain tumor survival through actin cytoskeleton reorganization

In this context, the production of ROS is generally active but maintained at a moderate level, a range often referred to as the pro-inflammatory threshold. At this concentration, ROS is sufficient to enhance immune cell activation without directly triggering apoptosis

Cysteine degradation catalyzed by CAT and MST (Figure 1B) is not deeply explored in cancer, since MST is more enzymatically efficient at a pH higher than the physiological, thus the role of CBS and CSE is considered more relevant in cancer biology (92)

PMID: 40921293 Exp Cell Res2025 Aug 25;452(1):114725