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Low to moderate levels of ROS contribute to sustaining cellular proliferation and can be a trigger for the activation of the stress-responsive survival pathway, including the glucose transporter 3,4

( 2 O 2 , a potent ROS-inducer, and superoxide dismutase (SOD), a natural antioxidant agent, were higher in GO then control OFs, whereas the ROS antagonist glutathione peroxidase (GPX) was reduced, with a reduced ratio between glutathione and oxidized glutathione, suggesting the presence of a remarkable state of oxidative stress ( Glycosaminoglycan Production and Heat Shock Protein 72 Expression in Orbital fibroblasts As mentioned above, the release of GAGs plays an important role in the pathogenesis of GO

and the time scales of animal studies (months) are incommensurable with the decades over which aging interventions would need to operate in humans

(Skin Barrier) 5