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Radiation-induced immunogenic modulation of tumor enhances antigen processing and calreticulin exposure, resulting in enhanced T-cell killing

Toxicol Sci 115: 513520

HCMECs at passages 10-15 were used for MARS1 knockdown experiments, while those at passages 3-8 were applied for MARS1 and/or HMGB1/2 overexpression studies

Second, MDA-MB-231 and BoM cells used a larger fraction of ATP for pathways other than nucleotide, fatty acid, and GSH synthesis (28 and 56%, respectively, vs