Our in vitro experiments indicated that Ent1 is a stronger binder than Ent2, and the FRET experiments showed that they target preferentially the Clathrin and Arrestin boxes, respectively
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We assessed keratin expression and found that the elevated expression of K16 and K17 diminished in both in vivo and in vitro experiments following SFN treatment
Whether it's the underlying cause or not clearly is a big question
