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GBM often exhibits resistance to conventional apoptosis-inducing therapies, and its immunosuppressive microenvironment limits the efficacy of existing treatments

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Several factors cause the sudden elevation of cellular contents, malondialdehyde (MDA) levels, and changes in enzyme activity, such as peroxidase (POD), catalase (CAT), and superoxide dismutase (SOD)

Thyroid hormones, particularly triiodothyronine (T3) and thyroxine (T4), are transported across the bloodbrain barrier (BBB) by specialized carriers and are metabolically activated or inactivated by local deiodinase enzymes (16)