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Acute myelosuppression occurs shortly after chemotherapy, while residual bone marrow injury manifested by a decrease in hematopoietic stem cell reserves or a myelodysplastic syndrome (Wang et al., 2006)

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Our findings demonstrate that DUX4-provoked toxicity is involved in the activation of the ferroptosis-related pathway and that supplementary iron could be a promising and readily available therapeutic option for FSHD