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Transfection of the GSTA1-1 gene into H69 cells (small-cell lung cancer) leads to resistance towards doxorubicin, exhibiting prooxidant action [101]

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An animal model found that giving d-alpha-tocopherol, a form of vitamin E, to rats 24 hours before a high dose of doxorubicin reduced cardiotoxicity without interfering with the chemotherapeutic effects of doxorubicin on leukemia (Sonneveld 1978)