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In our previous study, 50 mg of vitamin B-6 supplementation for 12 weeks could mediate antioxidant capacity by reducing plasma homocysteine in patients with hepatocellular carcinoma [14], in consequence, we used the same dosage (50 mg/d pyridoxine-HCl) to treat our patients in the present study

Figure 2 illustrates the pictorial representation of the targets unveiling quercetins interaction leading to a reduction in glucose levels

Several factors can contribute to premature greying, and one potential culprit is a deficiency in vitamin B12

The bioenergetic and antioxidant status of neurons is controlled by continuous degradation of a key glycolytic enzyme by Apc/C-Cdh1