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Since cisplatin-induced hearing loss was first reported 9 , numerous studies have investigated the physiological features and mechanisms of ototoxicity caused by cisplatin

There is extensive crosstalk between these cell death mechanisms, mainly through processes involving mitochondria, glutathione and the cystine transporter SLC7A11

Rosmarinic acid suppressed cisplatin-induced apoptosis by repressing ROS generation, cytochrome C release, caspase activation, AIF translocation, NF-B activation, IL-1 production, Bax upregulation, and Bcl-2 down-regulation, (Jeong et al

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