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In Table 1, we present a comparative analysis of other proteins involved in cancer treatment, particularly those influencing ER stress or ferroptosis

However, persistently elevated interferons and receptor activation of the MNK1/MNK2eIF4E signaling pathway at all disease phases caused sensorymotor dysfunction and pain by inducing hyperexcitability and sensitization of a GFRA3 + C-fiber subtype of joint-innervating sensory neurons

IL-6 appears to influence dramatically the nature of the immune response by dictating the recruitment, activation and apoptotic clearance of individual leucocyte subpopulations (e.g

The two approaches are shown below