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The specific mechanisms by which ferroptosis contributes to CHD pathophysiology require further elucidation, particularly regarding its interrelationship with other cell death pathways (e.g., apoptosis, necrosis)

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PPA caused a significant reduction in GABA levels while increasing glutamate excitotoxicity, mimicking the neural excitation dominance observed in ASD ( Histopathologically, PPA-treated rats exhibited hippocampal neurodegeneration, a crucial region for social and cognitive processing ( GABA supplementation provided moderate improvements in both neurotransmitter balance and oxidative stress markers

Peptides natural to the body were only available for a short time in the stomach, resulting in a lack of bioavailability when taken by mouth