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H 2 S causes vascular endothelial and smooth muscle cell hyperpolarization and vasorelaxation by activating K ATP channels through cysteine S-sulfhydration, which can enhance Kir6.1-PIP2 binding and reduce Kir6.1-ATP binding (19)

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For instance, if a patient switched from atorvastatin to rosuvastatin, both were explicitly recorded rather than collapsed under statin therapy. This ensured a precise representation of pharmacological history, enhancing the models ability to capture drug-disease interactions