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Pro-inflammatory cytokines and iron overload activate microglia and increase its polarization from the anti-inflammatory phenotype (M2) to the pro-inflammatory phenotype (M1), which aggravates pro-inflammatory cytokine production, including NO [306]

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Alternatively, if cannabinoids are able to reduce the opening of hemichannels during a chronic inflammatory event, it may subsequently prevent excitotoxicity caused by excess glutamate and reduce the release of pro-inflammatory cytokines, thus maintaining neuronal and astrocyte viability (Froger et al., 2009