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*Correspondence: Haohao Zhu [email protected] Zhou [email protected] Jiang [email protected] These authors have contributed equally to this work This article was submitted to Cellular Neuropathology, a section of the journal Frontiers in Cellular Neuroscience Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

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Increased translocation of Gram-negative bacteria with increased LPS load is probably a major trigger factor, but also other bacterial infections, toxoplasmosis, viral infections (e.g., cytomegalovirus), cancer, and gastrointestinal, autoimmune, immune-inflammatory, neuroinflammatory, and neurodegenerative disorders appear to be associated with those pathways via activation of TLR/LTF/TWEAK signaling