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RNA LINC00618 promotes apoptosis by increasing the levels of the pro-apoptotic protein, cleaved caspase-3, and also induces ferroptosis by inhibiting the cellular antioxidant system, suggesting a potential interaction between apoptosis and ferroptosis (168)

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Specifically, in our previous study, we determine if glutathione redox status could be improved with a three-month intervention of subcutaneously injected methylcobalamin and oral folinic acid in children with AD and metabolic evidence of abnormal methylation and glutathione metabolism [23]