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Reactive oxygen species (ROS) are produced by the mitochondrial ETC 50

In vitro data showed HLF inhibited the formation of foam cells by promoting cellular cholesterol efflux via the upregulation of the ATP-binding cassette transporter A1 (ABCA1), PPAR, and downregulation of CD36, thus preventing the progression of atherosclerotic lesions (Liu et al., 2009a)