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In AD, the identification of histone lactylation marks such as H4K12la and H3K18la links metabolic dysfunction to sustained microglial activation and inflammatory gene expression, integrating metabolic and epigenetic mechanisms underlying synaptic damage and cognitive decline (Pan et al., 2022

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Other previous experiments have shown that subcutaneous administration of rotenone resulted in a highly selective dopaminergic damage in neurons and -synuclein aggregation, similar to the Lewy bodies of PD [63,65]