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Figure 8 Discussion The present work illustrated a comprehensive workflow for pan-cancer analysis and thoroughly investigated the role of IGF-1 and IGF-1R in cancers

This apparent paradox arises from their context-dependent roles: cell death in normal intestinal cells may disrupt mucosal homeostasis, thereby aggravating inflammation and CD pathogenesis, whereas selective elimination of damaged cells via these pathways might suppress disease progression and synergize with other therapies