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Additionally, disulfide compounds such as disulfiram (DSF) and fursultiamine disrupt the GPX4-HSC70 interaction, inhibiting GPX4 degradation and enhancing ferroptosis resistance [84]

According to OBRA, the use of antidiabetic medications should include monitoring (e.g., periodic blood glucose) for effectiveness based on desired goals for that individual and to identify complications of treatment such as hypoglycemia or impaired renal function

The FSP1/CoQ10/NADPH system protects cells from ferroptosis in a manner independent of the GPX4 axis by inhibiting lipid peroxidation and ferroptosis through the NAD(P)H-dependent reduction of CoQ10 to ubiquinol (CoQH 2 ) [23]