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Copper chelation inhibits pivotal VSMC migration in intimal hyperplasia [194] improves recovery after myocardial infarction [222] and reduces neointimal formation and atherosclerosis in ApoE-deficient mice [223]

This action stimulates the release of growth hormone (GH) in a pulsatile manner a more physiological pattern than exogenous GH injections

Notably, the levels of OPA 1 mitochondrial dynamin like GTPase (OPA1) and mitofusin-2 (MFN2), which are crucial for mitochondrial fusion, were markedly decreased, suggesting impaired fusion processes

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