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Where can we develop therapies

Journal of Neuroimmune Pharmacology

Indeed, the defining features of trained immunity are reminiscent of aspects of HSC aging, such as myeloid-biased differentiation 31,32 and epigenetic reprogramming 34

These mice carry two mutations in the amyloid precursor protein gene, the Arctic (E693G) and Swedish (KM670/6701NL) mutations, and exhibit amyloid- deposits, a hallmark of AD (Lord et al