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These findings showed that OVAinduced barrier dysfunction is mediated by OSdriven Notch overactivation, which is counteracted by lowdose butyrate through modulation of this signaling pathway

Similarly, expression of GGT in tumors enables cells to maintain elevated levels of intracellular glutathione and to rapidly replenish glutathione during treatment with pro-oxidant anti-cancer therapy

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12 h) increases GSH levels, GSH/GSSG ratio, GPX activity, and cell viability in primary cortical neurons treated with OGD/R, with the strongest effect observed at 2 mM for 12 h [6]