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International Journal of Molecular Sciences [Internet]

Indeed, HGF protects BBB disruption after microsphere embolism-induced sustained cerebral ischemia by avoiding the inhibition of Bcl-2 (Date et al., 2006), thus decreasing apoptosis, and by reducing the expression of occludin and zonula occludens (ZO)-1, which are tight junction proteins, in cerebrovascular endothelial cells (Takeo et al., 2008)

which coat the surface of hydrophobic drug particles and render them hydrophilic

Regulatory Considerations Current Regulatory Status and Legal Framework The regulatory status of BPC-157 and TB-500 varies significantly across different jurisdictions and continues to evolve as regulatory agencies assess these compounds