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Although artemisinin leads to various tissue damages or molecular effects, causing more widespread changes than iron chelators, the ultimate effect of artemisinin or its derivatives is likely on the interference in iron utilization

These adjunctive inhibitors are likely to result in targeting both catalytic and cofactor binding sites, which will increase the binding affinity and selectivity of different DNMT isoforms

Under physiological conditions, Nrf2 is bound to its cytoplasmic inhibitor, Kelch-like ECH-associated protein 1(Keap1), and remains inactive

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