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Additionally, citrate-bound Mn (about 2% of plasma Mn) may enter the brain via a shared citrate transporter, as its influx exceeded diffusion and the transporter was competitively inhibited by Mn citrate ( Intracellularly, both DMT1 (Wolff et al., 2018) and mitochondrial calcium uniporter (Kamer et al., 2018) can participate in Mn 2+ uptake into the mitochondria in vitro (Figure 1D)

Contractile elements in the regulation of macromolecular permeability

Instead, it is possible that an ongoing process such as oxidation is responsible for the gradual dysfunction that manifests across myriad cellular pathways throughout the disease trajectory

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