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Key highlights include a systematic delineation of both core and emerging ferroptosis-regulatory networks, a detailed analysis of the pathological contributions of ferroptosis across distinct CVDs, and a critical evaluation of evolving therapeutic strategies ranging from small-molecule inhibitors to advanced nanomedicine-based approaches

Compared with untreated endometriosis mice, the expression levels of PCNA and VEGF were significantly reduced, however, E-cadherin was significantly increased in the nobiletin-treated group

Nrf2 is a key regulator of the constitutive or inducible molecular system that controls redox homeostasis, and its function may be inhibited in PD (da Costa Caiado et al., 2025)