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Mitochondrial dysfunction, manifested in the abnormal activities of the electron transport chain and impaired energy metabolism, greatly contributes to ASD

Research on the antisense oligonucleotide-based regulation of FMO3 in animal models revealed a decrease in TMAO serum levels concurrent with a reduction in diet-enhanced atherosclerosis (223)

Neither high (Figure 5, group 2) nor low levels (Figure 5, groups 5 and 7) of plasma medium and long chain acylcarnitines noted in our work correlate with described genetic disorders of mitochondrial fatty acid or organic acid metabolism in humans (64)

Brief Funct Genomics