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Retrieved 15 January 2017

This macrophage reprogramming is associated with SFN-mediated inhibition of the transcription factor c-Myc, which normally drives M2 polarization, and concurrent activation of the Nrf2 pathway that favors M1-associated gene expression profiles (78)

Furthermore, the infrequency of these connections in the current study is likely exaggerated by the use of very small tracer volumes injected into discrete cutaneous areas