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and finally, genetic predisposition is essential for the diseases development

Specifically focusing on empagliflozin, recent evidence demonstrates that this SGLT2 inhibitor directly improves endothelial function and reduces mitochondrial oxidative stress independent of glycemic control

Stabilization of GNAS was found to activate the PI3K/AKT signaling pathway, which was hypothesized to contribute to chemotherapeutic resistance via AKT-mediated DNA damage response mechanisms [63]