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The cellular abundance of iron, and hence the sensitivity to ferroptosis, is determined by the availability of ferritin, the level of transferrin, and the function of FPN [29, 30]

It is often referred to by its structure-based name, Modified GRF (1-29)

pro-oxidant therapy

In addition, compared to controls, CartIR / mice showed an increase in the number of PCNA-positive chondrocytes in the proliferating zone (Figure 2k and 2l) and a reduction in size of hypertrophic chondrocytes (Figure 2m and 2n)