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The hallmark pathological feature of AS is excessive cholesterol accumulation in arterial walls, leading to progressive thickening and hardening ( 2.1 Endothelial dysfunction and inflammatory regulation Atherosclerosis can be broadly divided into several stages: lipid deposition, persistent inflammatory response, foam cell formation, vascular smooth muscle cell (VSMC) proliferation, and VR ( B activation and reducing the release of pro-inflammatory cytokines ( 2.2 Lipid deposition and foam cell formation Following endothelial cell injury, structural alterations and functional impairment lead to a marked increase in vascular permeability, facilitating the penetration of large amounts of LDL into the subendothelial space

The potential role of infectious agents in AD pathogenesis has gained increasing attention 2,3,4,5 , with Chlamydia pneumoniae , an obligate gram-negative bacterium primarily responsible for community-acquired pneumonia, emerging as a significant pathogen 6,7,8

What causes low levels of vitamin B12

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