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Combined these data provide novel insight into the therapeutic actions of HGF/MET activation to ameliorate cognitive deficits after repeated mTBI

Considering this, CVB infection, particularly persistent infection, may contribute to the overall demise of the cell population by further activating autoreactive T cells, augmenting the effects of autoimmunity, and by further inducing cell stress and death

The downstream consequences include enhanced glucose uptake into skeletal muscle independent of insulin, increased fatty acid oxidation, and suppression of inflammatory signaling

This article shows that activation of hypothalamic GIPR cells/neurons decreases food intake