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Abstract The mechanisms underlying how cells subjected to genotoxic stress reestablish reduction-oxidation (redox) homeostasis to scavenge genotoxic stress-induced reactive oxygen species (ROS), which maintains the physiological function of cellular processes and cell survival, remain unclear

Animals Hyh mutant mice (B6C3Fe a/a-Napa hyh /J) carrying the M105I mutation were obtained from The Jackson Laboratory (Bar Harbor, USA) and maintained at Universidad Austral de Chile (Valdivia, Chile)

Rings showing 80% or greater relaxation were considered to have intact endothelium

In contrast, nanozymes have emerged as a promising non-enzymatic alternative due to their superior stability