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By encapsulating chemotherapy drugs and gene therapy agents (e.g., siRNA, miRNA, or plasmid DNA) in nanocarriers, synergistic effects can be achieved

These approaches quantify E/I ratios using biomarkers such as the Hurst exponent (H), derived from resting-state fMRI or EEG data, which correlates with the long-range temporal correlation of neural activity

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In addition to the PERK-ATF4-CHOP axis, other key ER stress pathways, such as IRE1/XBP1 and ATF6 also contribute to the UPR, collectively managing protein misfolding and restoring homeostasis, or, if stress persists, trigger cell death (Tabas and Ron, 2011)