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For instance, PKC ) or overexpression of a cleavage-resistant PKC mutant protected against METH-elicited apoptosis in mesencephalic dopaminergic cell cultures in vitro ( PKC inhibition may rescue METH-elicited mitochondrial burden, pro-apoptosis, and dopaminergic degeneration, implying that PKC is an important gene involved in METH-induced mitochondrial dysfunction and apoptosis in dopaminergic neuronal cells ( Recently, Zhang et al

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