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Empirical therapy for mild CAP often comprises a macrolide, in locations with high rates of macrolide resistance, particularly in patients with comorbidities, a respiratory fluoroquinolone or a combination of a beta-lactam and a macrolide is suggested [21]

Hydrogen sulfide in paraventricular nucleus attenuates blood pressure by regulating oxidative stress and inflammatory cytokines in high salt-induced hypertension

However, their dysregulation can exacerbate inflammation and tissue damage in ARDS ( Given the central role of oxidative stress and lipid peroxidation in ARDS, targeting ferroptosis represents a promising therapeutic strategy ( 2 Ferroptosis in infection and ARDS development Cell death pathways play critical roles in immune homeostasis, infection defense, and inflammation resolution

Malaguarnera M, Pistone G, Elvira R, et al