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showed that V-B12, but not its CN-bound analogue (cyanocobalamin), was responsible for the increase in arterial blood pressure.6 The loss of the hypertensive effect of V-B12 by the non-selective NOS inhibitor L-N-nitro-L-arginine methyl ester further corroborates the finding that NO scavenging is a mechanism for elevating blood pressure.10 Vitamin B12 has also been suggested to interfere with the production of NO

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