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Alpha-1 Antitrypsin Deficiency and Oxidative Stress Among individuals with COPD, those with alpha-1 antitrypsin deficiency have a distinct pattern of oxidative stress characterized by an increase in exhaled breath nitric oxide, accelerated polymerization of oxidized Z-AT (Glu342Lys) protein which increases inflammation, and decreased serum glutathione and catalase before the appearance of COPD

The intracellular GSH is decreased with the increased CCF concentration, accompanied by increased tumor cell apoptosis (Figure 5B), which demonstrates the antitumor effect of GSH-promoted CDT

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What cooking methods best preserve glutathione in foods